Obesity

Obesity is a common, serious, complex disease particularly associated with the modern way of living. This medical condition is strongly associated with an increased risk for heart disease, type 2 diabetes and various types of cancer. Obesity usually comes as a result of unhealthy way of living, poor nutrition, lack of physical exercise, inherited factors and/or other environmental factors.

Your Health Specialist will address a series of targeted questions to you, to assess whether your condition can be characterized as Obesity.

Weight Loss with Molecular Nutrition

• Ultimate Weight Loss (Weight Conservation)
• Molecular Nutrition with Doctor Supervision
• Specialized Tests – Full Metabolic Profile Test
• Energy Increase at optimal levels
• Addressing the causes that cause weight gain at a cellular level

Then your specialist may recommend specialized blood tests or other molecular-level diagnostic tests to identify possible hormonal and/or biochemical imbalances. In some cases a consultation with a Cardiologist may be recommended. Your personalized treatment plan will be based upon the diagnostic tests results, the symptoms described and your medical history overall.

This treatment plan will be accompanied by a Molecular Nutrition program that will be compiled after a distinct consultation with our Molecular Nutritionist and will aim towards achieving even higher levels of health and well-being along with losing weight and alleviating the true causes of Obesity.

References

• Centers for Disease Control and Prevention. Second National Report on Human Exposure to Environmental Chemicals, NCEH Publication No. 03-0022. Atlanta, GA: Centers for Disease Control.
• Pelletier C, Imbeault P, Tremblay A. Energy balance and pollution by organochlorines and polychlorinated biphenyls. Obes Rev. 2003;4(1):17-24.
• Evans JL, Goldfine ID, Maddux BA, Grodsky GM. Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes. Endocrine Reviews 23(5):599–622.
• Girard D. Activation of human polymorphonuclear neutrophils by environmental contaminants. Rev Environ Health 2003;18(2):75-89.
• Sonnenberg GE, Krakower GR, Kissebah AH. A novel pathway to the manifestations of metabolic syndrome. Obes Res. 2004;12(2):180-186.
• Wang Z, Zhou YT, Kakuma T, et al. Leptin resistance of adipocytes in obesity: role of suppressors of cytokine signaling. Biochem Biophys Res Commun. 2000;277 (1):20-26.
• Beattie JH, Wood AM, Newman AM, Bremner I, et al. Obesity and hyperleptinemia in metallothionein (-I and -II) null mice. Proc Natl Acad Sci U S A. 1998;95(1):358-363.
• Goto M, Arima H, Watanabe M, et al. Ghrelin increases neuropeptide y and agoutirelated peptide gene expression in the arcuate nucleus in rat hypothalamic organotypic] cultures. Endocrinol. 2006.;147(11):5102-5109.
• Wynne K, Stanley S, McGowan B, Bloom S. Appetite control. J Endocrinol. 2005;184(2):291-318.
• Dube MG, Pu S, Kalra SP, et al. Melanocortin signaling is decreased during neurotoxin-induced transient hyperphagia and increased body-weight gain. Peptides. 2000;21(6):793-801.
• Shoemaker RC, House DE. A time-series study of sick building syndrome: chronic, biotoxin-associated illness from exposure to water-damaged buildings. Neurotoxicol Teratol. 2005;27(1):29-46.
• Catania A, Delgado R, Airaghi L, et. al. alpha-MSH in systemic inflammation. Central and peripheral actions. Ann N Y Acad Sci. 1999;885:183-187.
• Dube MG, Pu S, Kalra SP, et al. Melanocortin signaling is decreased during neurotoxin-induced transient hyperphagia and increased body-weight gain. Peptides. 2000;21(6):793-801.
• Shoemaker RC, House DE. A time-series study of sick building syndrome: chronic, biotoxin-associated illness from exposure to water-damaged buildings. Neurotoxicol Teratol. 2005;27(1):29-46.
• Gladen BC, Ragan NB, Rogan WJ. Pubertal growth and development and prenatal and lactational exposure to polychlorinated biphenyls and dichlorodiphenyl dichloroethene. J Pediatr. 2000;136(4):490-496.
• Tremblay A, Pelletier C, Doucet E, et al. Thermogenesis and weight loss in obese individuals: a primary association with organochlorine pollution. Int J Obes Relat Metab Disord. 2004;28(7):936-939.
• Imbeault P, Tremblay A, Simoneau JA, et al. Weight loss-induced rise in plasma pollutant is associated with reduced skeletal muscle oxidative capacity. Am J Physiol Endocrinol Metab. 2002;282(3):E574-E579.
• Nicolau GY, Pesticide effects upon the circadian time structure in the thyroid, adrenal and testis in rats. Endocrinologie. 1982;20(2):73-90.
• Duke University Integrated Toxicology Program, National Institute of Environmental Health Sciences/NIH/DHHS, Obesity: Developmental Origins and Environmental Influences. Available at qt/dert/obesity/agenda.htm.
• Moskaug JO, Carlsen H, Myhrstad MD, et al. Polyphenols and glutathione synthesis regulation. Am J Clin Nutr. 2005;81(1 Suppl):277S-283S.
• Masuda A, Miyata M, Kihara T, et al. Repeated sauna therapy reduces urinary 8-epiprostaglandin F(2alpha). Jpn Heart J. 2004;45(2):297-303.
• Biro S, Masuda A, Kihara T, Tei C. Clinical implications of thermal therapy in lifestyle-related diseases. Exp Biol Med (Maywood). 2003;228(10):1245-1249.
• Hyman M. Ultrametabolism. New York: Scribner; 2006.
• Hyman MA. Systems biology: the gut-brain-fat cell connection and obesity. Altern Ther Health Med. 2006;12(1):10-16
• Hyman MA. Refrigerator rights–the missing link in health, disease, and obesity, Altern Ther Health Med. 2005;11(4):10-12.
• Lyon M, Bland J, Jones DS. Chapter 31: Clinical approaches to detoxification and biotransformation. In: Jones, DS (Ed). Textbook of Functional Medicine. Gig Harbor, WA: Institute for Functional Medicine; 2006.